Neutrophils are the most abundant leukocytes in human circulation and play central roles in host defense and tissue homeostasis. My research has long been driven by a fascination with how these highly dynamic cells adapt and respond across diverse physiological and pathological settings. During my PhD study at the Weizmann Institute of Science, I investigated how inflammatory bone marrow neutrophils reprogram their metabolism during infection and inflammation, and how lactate release regulates their mobilization. More recently, during my postdoctoral research at the University of Cambridge, I uncovered a novel mechanism by which neutrophils evade clearance through the “don't eat me" signal CD24, thereby promoting pathological interactions with megakaryocytes through unique cell-in-cell process of emperipolesis and contributing to disease progression in blood cancers.​

In my lab, we investigate how neutrophil plasticity, ageing, metabolism, and interactions with the surrounding microenvironment shape disease initiation and severe complications. We are particularly interested in understanding the roles of neutrophils in sepsis, infectious diseases, and blood cancers. Using human and preclinical models together with cutting-edge techniques, we aim to bridge fundamental mechanisms with translational insight and uncover new opportunities for early diagnosis and therapeutic intervention.​​​